Alzheimer's Disease

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 I understand that Alzheimer's is an  irreversible disease, a progressive brain disorder. I am a student of electrical engineering. In electrical engineering science, with a proper feedback system, things can be reversible.  I feel that we need more research on this  Alzheimers. I think we should learn to use any particular method to make it reversible! It allows you the flexibility to experiment and test your concept at any time while maintaining control over how it becomes reality. This could allow for something truly life-changing.  Alzheimer's is irreversible? - "Yes it is". You can stop taking them, but you cannot prevent people from getting Alzheimer's." Treatments for Alzheimer's are based on trial and error. Dr. George Lacey says treatments like the Tylenol Prodrug - a pharmaceutical that works by reducing inflammation in your blood cells while also removing plaque buildup around brain tissue, to slow down or even reverse the deterioration of AD c

 I was remembering it a German this is good but it might come but then those could be sick because I don't think about America what about the discrepancy Buxtehude but a man do you do we do have are causing some stuff but there was a lousy all up getting post much department loaded the local important that's so good to go but they can't lock up put it in all it was like it was going home is a  keep up cool publicly bonus ability to quickly get you could join up what could you react easily doing it would you look anywhere it was just that and if you want to watch because we knew that he got in the popular G. or with windows 8 you can get some looks we look but by using this link so now it is because of all the topic.  I am not disease what can look again what does this keep this going memory disorders V. both sustenance okay yes this is musically up with him and what he had to learning I'm learning about what head do okay unison capacity Hey yup joining ever to be awarde

  A century ago a German doctor called Alice outside spotted anomalies in brain sections from a patient with dementia.  Ever since people have been studying the strange plaques and tangles he saw in the hope that we could one day on the stand and kill what is now known outside his disease.  Insoluble deposits of a peptide called amyloid-beta A. B.   The phone when approaching.  Because the protein is sequentially clean by 2 and a beta and gamma secretase.  All the molecules are generated by this cleavage and may play a role in the disease but A. B. tech is the main culprit.  ABC tends to miss full become sticky eventually clumping together home saludable all the goodness.  Some of these aggregates into large insoluble fibrils the deposits in the brain plaques.  The.  In several forms of speech.  We don't know exactly which species is toxic research shows that the communication and plasticity sign up.  This could be what stops the brain from full.  The big memories.  New homes on th

Welcome to this awesome brand have video on outside his disease with the aid of some diagrams I'm going to describe the pathogenesis about Simon's resulting microscopic and macroscopic changes which take place and some of the clinical features which manifest with these changes.  Sir what is awesome is disease Alzheimer's disease is a chronic nerve degenerative disease of the cortex which begins insidiously as impairment of high cognitive function and progresses resulting deficits of memory visuospatial orientation judgment personality and language these changes typically occur over a 5 to 10 year.   First let's talk about the functioning of the house in your own in the part of the cell membrane of neurons which forms a sign ups there are numerous proteins called amyloid precursor proteins otherwise known as A. P. P. and as with all proteins old versions of a PPO degraded and new ones take their place 8 PP is normally broken down into soluble peptide spine enzyme called

  Time is deceased was described for the first time in 1907 by the German psychiatrist Eloise as a timer.  In performing histopathologic studies of the brain of his patient Augustine suffering from dementia he brought to light the presence of 2 types of lesions in the brain.  Senile plaques and neurofibrillary tangles he reached the conclusion of a distinct disease of a cerebral cortex 100 years later thanks to current scientific techniques research has made a great leap and the understanding of the disease.  We know that the brain is made up of neurons and that these are interconnected to form a vast network.  These connections known as synapses enable the transmission of information from one neuron to another.  And now it's time for us to cease 10 to 15 years before the appearance of the symptoms to main lesions forms in the brain.  Senile plaques composed of amyloid-beta protein and neurofibrillary tangles composed of tau protein.  How is the senile plaque formed?  On the surfac