Alzheimer's Disease

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  Time is deceased was described for the first time in 1907 by the German psychiatrist Eloise as a timer.  In performing histopathologic studies of the brain of his patient Augustine suffering from dementia he brought to light the presence of 2 types of lesions in the brain.  Senile plaques and neurofibrillary tangles he reached the conclusion of a distinct disease of a cerebral cortex 100 years later thanks to current scientific techniques research has made a great leap and the understanding of the disease.  We know that the brain is made up of neurons and that these are interconnected to form a vast network.  These connections known as synapses enable the transmission of information from one neuron to another.  And now it's time for us to cease 10 to 15 years before the appearance of the symptoms to main lesions forms in the brain.  Senile plaques composed of amyloid-beta protein and neurofibrillary tangles composed of tau protein.  How is the senile plaque formed?  On the surfac

  What is Alzheimer's disease?  Alzheimer's is a slow fatal disease of the brain affecting one in 10 people over the age of 65.  No one is immune.  The disease comes on gradually as abnormal protein fragments called plaques and tangles accumulate in the brain and kill brain cells.  They start here in the hippocampus the part of the brain where memories are the first to form.  Over many years' time, the plaques and tangles slowly destroy the hippocampus and it becomes harder and harder to form new memories.  Simple recollections from a few hours or days ago but the rest of us might take for granted are just not there.  After that more plaques and tangles spread into different regions of the brain.  Ellen cells and compromising function wherever they go this is spreading around is what causes the different stages of Alzheimer's.  From the hippocampus, the disease spreads here to the region of the brain where the language is processed when that happens it gets tougher and